Mon Jun 8, 2015 6:52 am (PDT) . Posted by:
"Rick Laferriere" ri_lymeinfo
Abundance and infection rates of Ixodes scapularis nymphs collected from residential properties in Lyme disease-endemic areas of Connecticut, Maryland, and New York
Katherine A. Feldman, Neeta P. Connally, Andrias Hojgaard, Erin H. Jones, Jennifer L. White and Alison F. Hinckley
Journal of Vector Ecology, Volume 40, Issue 1, pages 198–201, June 2015
http://doi.org/10.1111/jvec.12153 http://doi.org/10.1111/jvec.12153
Ixodes scapularis, commonly known as the blacklegged tick, is responsible for transmitting Lyme disease (caused by Borrelia burgdorferi), the most common vector-borne disease in the United States (Centers for Disease Control and Prevention 2014). The blacklegged tick can also transmit Anaplasma phagocytophilum (the etiologic agent of human granulocytic anaplasmosis), Babesia microti (the causative agent of babesiosis), Borrelia miyamotoi (a relapsing fever Borrelia), and deer tick virus. In the northeastern U.S., the highest risk of exposure to the blacklegged tick is likely peridomestic, due to fragmented forest landscapes and other land-use characteristics, as well as the intrusion of humans into prime habitat for blacklegged ticks and their hosts. Despite this, most reports of tick abundance and infection rates focus primarily on ticks collected from public lands and forested research sites.
We collected ticks from residential properties in Lyme disease-endemic areas and determined infection rates for nymphal I. scapularis as part of a two-year, multi-site tickborne disease intervention study involving the Centers for Disease Control and Prevention (CDC) and the Emerging Infections Programs in Connecticut (CT), Maryland (MD) and New York (NY). Here, we present tick densities and infection rates for B. burgdorferi, A. phagocytophilum, and B. microti from nymphal I. scapularis, reflecting peridomestic exposure to these pathogens.
http://doi.org/10.1111/jvec.12153 http://doi.org/10.1111/jvec.12153
Free, full text (pdf file, 207 KB):
http://onlinelibrary.wiley.com/doi/10.1111/jvec.12153/epdf http://onlinelibrary.wiley.com/doi/10.1111/jvec.12153/epdf
Katherine A. Feldman, Neeta P. Connally, Andrias Hojgaard, Erin H. Jones, Jennifer L. White and Alison F. Hinckley
Journal of Vector Ecology, Volume 40, Issue 1, pages 198–201, June 2015
http://doi.org/10.1111/jvec.12153 http://doi.org/10.1111/jvec.12153
Ixodes scapularis, commonly known as the blacklegged tick, is responsible for transmitting Lyme disease (caused by Borrelia burgdorferi), the most common vector-borne disease in the United States (Centers for Disease Control and Prevention 2014). The blacklegged tick can also transmit Anaplasma phagocytophilum (the etiologic agent of human granulocytic anaplasmosis), Babesia microti (the causative agent of babesiosis), Borrelia miyamotoi (a relapsing fever Borrelia), and deer tick virus. In the northeastern U.S., the highest risk of exposure to the blacklegged tick is likely peridomestic, due to fragmented forest landscapes and other land-use characteristics, as well as the intrusion of humans into prime habitat for blacklegged ticks and their hosts. Despite this, most reports of tick abundance and infection rates focus primarily on ticks collected from public lands and forested research sites.
We collected ticks from residential properties in Lyme disease-endemic areas and determined infection rates for nymphal I. scapularis as part of a two-year, multi-site tickborne disease intervention study involving the Centers for Disease Control and Prevention (CDC) and the Emerging Infections Programs in Connecticut (CT), Maryland (MD) and New York (NY). Here, we present tick densities and infection rates for B. burgdorferi, A. phagocytophilum, and B. microti from nymphal I. scapularis, reflecting peridomestic exposure to these pathogens.
http://doi.org/10.1111/jvec.12153 http://doi.org/10.1111/jvec.12153
Free, full text (pdf file, 207 KB):
http://onlinelibrary.wiley.com/doi/10.1111/jvec.12153/epdf http://onlinelibrary.wiley.com/doi/10.1111/jvec.12153/epdf
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