Wednesday, February 29, 2012

Fish Oil Protects Against Diseases Like Parkinson's

ScienceDaily (Apr. 19, 2009) — Dr. Nicolas Bazan, Director of the Neuroscience Center of Excellence, Boyd Professor, and Ernest C. and Yvette C. Villere Chair of Retinal Degenerative Diseases Research at LSU Health Sciences Center New Orleans, will present new research findings showing that an omega three fatty acid in the diet protects brain cells by preventing the misfolding of a protein resulting from a gene mutation in neurodegenerative diseases like Parkinson's and Huntington's.
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Friday, February 24, 2012

PANDAS or Lyme - Which is it?

Researchers Publish Long Awaited Lyme Disease and PANDAS Review Despite Controversies

SAN DIEGO, Feb. 22, 2012 /PRNewswire-iReach/ -- Neurologic tics and obsessive-compulsive behaviors caused by a bacterial infection in children were once thought to occur exclusively in PANDAS (Pediatric Autoimmune Disease Associated with Streptococcus infections). Despite ongoing controversies regarding Lyme disease and PANDAS, a recent evidence-based review by Daniel J. Cameron, MD, MPH and Hanna Rhee, MD uncovered a little known published report of a child who initially appeared to have PANDAS, but was later found to have Lyme disease. When the patient was treated with antibiotics for the bacterial infection transmitted by a tick bite, his symptoms resolved.

"Our extensive review of the literature has elucidated the mechanism with which a tick bite or sore throat may develop into neuropsychiatric disorders. TLRs (Toll-like receptors) in haplotype-variable individuals may play a role in multisystemic disease formation," states Dr. Rhee.

"We chose to publish our review paper in the International Journal of General Medicine, an online publication which provides open access to the public so that families and their physicians can download a free copy to reference at their convenience. In the context of psychiatric pathology, maintaining a differential diagnosis inclusive of an infectious etiology is what may help to save that child's life," reports co-author Dr. Cameron.

Media Contact: Hanna Rhee Hanna Rhee, MD Inc, 760-814-9229, hannarhee@...

SOURCE Hanna Rhee, MD Inc.

Thursday, February 23, 2012

Friday, February 17, 2012

Borrelia persists after antbiotic treatment in Monkeys

A new study by Drs. Monica Embers, Stephen Barthold and colleagues has found that the bacteria that cause Lyme disease, Borrelia burgdorferi (Bb) persist in monkeys after antibiotic treatment. It is the latest in a number of studies that have demonstrated persistent infection in animal models despite treatment.

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Friday, February 10, 2012

Researchers discover how mutations in parkin gene cause PD

Structure of the PARK2 protein. Based on PyMOL rendering of PDB

Parkinson's disease researchers at the University at Buffalo have discovered how mutations in the parkin gene cause the disease, which afflicts at least 500,000 Americans and for which there is no cure.

The results are published in the current issue of Nature Communications. The UB findings reveal potential new drug targets for the disease as well as a screening platform for discovering new treatments that might mimic the protective functions of parkin. UB has applied for patent protection on the screening platform.

"This is the first time that human dopamine neurons have ever been generated from Parkinson's disease patients with parkin mutations," says Jian Feng, PhD, professor of physiology and biophysics in the UB School of Medicine and Biomedical Sciences and the study's lead author. ...

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Thursday, February 9, 2012

Tai Chi helps with Parkinson's disease

Tai Chi may improve Parkinson's symptoms: research

An hour of Tai Chi twice a week can improve stability and walking ability in people with Parkinson's disease, a study has found.

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Wednesday, February 8, 2012

Genetic Parkinson's disease brain cells made in lab

By Michelle Roberts
Health reporter, BBC News

Abnormal accumulations of protein develop inside nerve cells in Parkinson's disease

Parkinson's research 'breakthrough'
Scientists in the US have successfully made human brain cells in the lab that are an exact replica of genetically caused Parkinson's disease.

The breakthrough means they can now see exactly how mutations in the parkin gene cause the disease in an estimated one in 10 patients with Parkinson's.

And it offers a realistic model to test new treatments on - a hurdle that has blighted research efforts until now.

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Sunday, February 5, 2012

Dr. Teitelbaum's Lyme and chronic disease regimen

Chronic Fatigue and Fibromyalgia Expert Creates Comprehensive Recovery Program That Truly SHINEs
Here's an article by Scott Forsgren (also known as The better health guy) about a recovery program for CFS and FM that you might find interesting. The treatment protocol applies to Lyme as well. It uses the acronym SHINE to help you remember the treatment plan. Lots of useful information in the article and the interview Q and A with the doctor.

Dr. Dietrich Klinghardt on Lyme Disease

Here's an interview with Seattle-based German doctor, Dietrich Klinghardt, about his unconventional treatments for Lyme, including reduction of EMF's, dealing with emotiomal issues, multi-generational family constellation issues, and more.

Saturday, February 4, 2012

What are IgG and IgM?

IgM vs IgG

An immunoglobulin or antibody refers to proteins that bind to antigens in specific cases. Both IgM and IgG refer to a class of immunoglobulin. Antibodies are produced by the immune system to fight antigens like bacteria and viruses. IgM refers to those antibodies that are produced immediately after an exposure to the disease, while IgG refers to a later response. IgG generally confers immunity to a patient so far as that particular disease is concerned.

There are a different immune responses to different antigens or ‘enemies’ that threaten the body. For instance, the antibody produced by your body in response to an exposure to chicken pox is different from the response it produces in case of mononucleosis. At times, the body may mistakenly produce antibodies even against itself! This results in an autoimmune disease.

Immunoglobulin G or IgG is the antibody that is found most abundantly in the human body. It is found in all bodily fluids and it protects the human body against bacterial and viral attacks.

Immunoglobulin M on the other hand is found mainly in the lymph fluid and in blood. This is the first antibody that is produced by the human fetus. It is also the first antibody that is produced in case of an exposure to a particular disease.

IgG and IgM are usually measured together by your doctor when you go in for a test. When they are evaluated together, they give your doctor a better idea about the functioning of your immune system.

An important difference between the two antibodies is related to exposure. While IgM antibodies are usually found in a human body after it has been exposed to a disease, IgG is the long term response of the body to a disease. For instance, if a kid is exposed to chickenpox, he will show elevated results of IgM in the blood in the period following the exposure. Once the child gets the disease, he acquires long term immunity against it by developing IgG antibodies. While IgM is an indicator of a current infection, an IgG indicates a recent or past exposure to the illness.

IgM is the first antibody that the body produces in an acute infection. It is approximately six times larger than IgG and is multivalent. This means that it has multiple binding sites. In the case of IgM, the number of binding sites is 10! However, only about half of these can actually bind to an antigen.

IgM is a temporary antibody that disappears within two or three weeks. It is then replaced by IgG which lasts for life and provides lasting immunity to the person.


1. IgM is the immediate antibody that is produced once a human body is exposed to a bacteria, virus or a toxin

2. IgG is found throughout the body, mainly in most of the bodily fluids, while IgM is found mainly in the blood and lymphatic fluids.

3. IgM is larger in size compared to IgG

4. IgM is temporary and disappears after a few weeks. It is then replaced by IgG.

Read more: Difference Between IgM and IgG | Difference Between | IgM vs IgG

Thursday, February 2, 2012

Essentiale-N IV’s


The work of Patricia Kane PhD has brought to light the crucial importance of phosphatidylcholine (PC). Perhaps you have never heard of it. It is a component of lecithin, and the major phospholipid of every human cell membrane. It is also a precursor to acetylcholine, a major neurotransmitter, via its intermediary choline. It is used in orthodox medicine for the treatment and prophylaxis of fatty embolism, and is the active ingredient in Plaque-X which has been marketed and used In Europe for the treatment of arteriosclerosis and reversal of plaque for many years. Drs Kane and Braccia have developed their own protocol for administration of the imported Essential- N brand of PC and have been applying it clinically for neurodetoxification and for the treatment of neurodegenerative disorders such as Parkinson’s, MS, and ALS, as well as Lyme, CFIDS, fibromyalgia, autism, hepatitis, and mood disorders. The “PK Protocol” named after originator Patricia Kane PhD, involves IV push of lower doses of stabilized PC known as Essential –N. This is the product Caring Medical now uses for all therapeutic administration of PC. Certain neurologic conditions will require twice daily injections of PC, often followed by IV glutathione, but the maximum amount of Essentiale-N per IVP is 2 vials or 10cc given up to twice daily. The usual frequency of IV’s is twice weekly. Successful therapy with PC is associated with a drop in serum CPK. and improvement in the FACT vision testing score. The IV is administered as a short IV push. ...

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