Thirty-four-year-old Chris Valerio is a prisoner in his own body. Just standing up is an ordeal. He can't feed himself or talk much, but his parents say his mind is fine, he understands everything.
"It's very frustrating. Obviously it's very disheartening. We've had our days of tears," said John Valerio, Chris' father.
For his parents, it's heartbreaking to see Chris, who'd been such a free spirit. He loved to travel and try new restaurants. Now he can't chew, or do anything for himself.
http://medicalxpress.com/news/2014-07-link-lyme-alzheimer-diseases.html
Journal of Alzheimer's Disease, May 19, 2014. DOI: 10.3233/JAD-140552
Collexis Holdings, Inc.
Life Science Solutions Leader
Europe, Via Bramantino 1, 6600
Locarno, Switzerland
Corresponding author: Aaron A. Sorensen
E-mail: sorensen@collexis.com
Dear Aaron Sorensen,
In reference to the study disproving Lyme and Alzheimer's disease with its so-called "Author-disambiguation algorithms and bibliometric analysis platforms" where is the microscopic and biochemical evidence for this conclusion?
The Barry Marshall affect on chronic disease
Excerpt from the link above: (Must read entire link when you have a moment)
Twenty five years before researchers Barry Marshall and Robin Warren discovered H. pylori was causing peptic ulcer disease (not stress or diet) a physician in Greece was successfully treating with antibiotics.
John Lykoudis, a general practitioner in Greece reportedly treated more than 30,000 patients.
Now imagine if you will that a bacterium was responsible for the chronic diseases of our time; Multiple Sclerosis, Lupus, Lou Gehrig's, disease, Parkinson's, Alzheimer's disease, or Fibromyalgia? How large is the existing revenue stream for treating these diseases? Calculating exact numbers would be insurmountable.
Ironically these are the same diseases that Lyme patients are routinely misdiagnosed with as we hear the stories over and over again at support group meetings. If Lyme patients don't obtain the correct diagnosis then their "misdiagnosis" is treated maintaining the status quo which would be motivation for refusing long-term antibiotic treatment.
Lykoudis was unable to persuade the Greek medical establishment regarding efficacy of his treatment and was given a fine of 4000 drachmas by a disciplinary committee, and indicted in the Greek courts while unable to get drug companies interested in treatment.
Like Marshall and Warren, Lykoudis found physicians resist challenges to prevailing medical dogma. Ulcers were such a cash cow for gastroenterologists and surgeons that the Mayo Clinic for example was built on gastric surgery. Surgeons and drug companies had a vested interest in maintaining the status quo. That revenue stream however would soon dry up.
History repeats itself.
If late stage untreated Lyme represents say only 25% of today's chronic illnesses it still presents a substantial threat to existing revenue streams. It is blatantly obvious that what has been put in place has been done so to deny the chronic or late stage Lyme epidemic.
http://article.sapub.org/10.5923.j.microbiology.20130306.11.html#Sec5
Journal of Microbiology Research
doi:10.5923/j.microbiology.20130306.11
Holly Ahern Biology Department, State University of New York at Adirondack, Queensbury, NY, 12804, USA
In this study 16% of respondents experienced cardiac, rheumatologic, or neurological symptoms consistent with late-stage Lyme disease. Comparing late-stage Lyme symptoms to the general population was a brilliant idea because we believe the vast majority of Lyme patients are already in the healthcare system being treated for everything else but Lyme.
Our current 2-tier Lyme disease analysis has a sensitivity of 59% which is not much better than a coin toss. Faulty diagnostic tests have led to the disability of tens of thousands of Lyme patients through misdiagnosis and delayed treatment. Late stage Lyme disease does not respond well to short courses of antibiotics leaving these patients with the dilemma and financial burden of finding a way to treat beyond the IDSA's one-size-fits-all treatment guideline. It is time to recognize that serodiagnostics for Lyme disease is inherently flawed and should be scrapped for testing capable of measuring treatment failure or success in all stages of disease. (Unless of course it is necessary to hide evidence of persistent infection for some undisclosed purpose.)
Author-disambiguation algorithms and bibliometric analysis platforms are not a substitute for accurate/reliable diagnostic tests.
Respectfully Submitted,
Carl Tuttle
33 David Dr
Hudson, NH 03051
Petition: Calling for a Congressional investigation of the CDC, IDSA and ALDF (22,431 signatures and growing)
Letter to the Editor, The Lancet Infectious Diseases Published May 2012
The Tuttle family was featured on New Hampshire Chronicle's "Living with Lyme" with the program archived on their site in six small segments for viewing on the computer
A two-year study out of Johns Hopkins (2005) concluded that laboratory tests for Lyme disease were only 50% accurate at best.
Source:
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1248466/
For physicians and researchers alike, fecal transplants present an opportunity to gain insight into disease
| November 3, 2010
Most people might find the idea of having another person's feces injected into their intestine hard to stomach, but for those with intractable gastrointestinal problems, another person's bodily waste is all that's standing between a lifetime of severe illness and a full recovery.
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This therapy—known as fecal transplants, bacteriotherapy, or human probiotic infusions—has taken to the limelight in recent years, not only because its gross factor makes for great headlines, but in great part because of the growing epidemic of a particularly toxic strain of Clostridium difficile that has been plaguing hospitals across the U.S. for the past decade and affecting more than a quarter of a million Americans per year. By producing sturdy spores that can linger in the intestinal tract even after repeated antibiotic treatment, C. difficile can continually give rise to new toxin-producing colonies that wreak havoc on the colon.
But these colonies prove no match for fecal transplants, which boast a cure rate of up to 95 percent. At the heart of these transplants are the trillions of microbes that inhabit the gut and have a profound impact on the host's biology—for better or worse. As Australian gastroenterologist, Thomas Borody, jokingly puts it, "we are 10 percent human, 90 percent poo."
Borody did his first fecal transplant back in the mid 1980s, when he was confronted with one of the most difficult cases he had seen at the time: a woman who had vacationed at Fiji and had developed an incurable colitis through an unknown pathogen. While searching the literature for alternative treatments, he stumbled upon a paper published in 1958 in the journal Surgery that described four cases in which a similar condition was cured by infusing the inflamed guts of the patients with fecal samples from healthy donors. "So I looked at the method and I kind of made up the rest of it," Borody said.
We compared the antibodies to B. burgdorferi in three different populations in order to evaluate the diagnostic reliability of Lyme borreliosis serologic analysis. The subjects included 25 patients with Lyme borreliosis (Group 1); 50 patients with diseases of unknown cause, B. burgdorferi ELISA-positive in serum and without B. burgdorferi infection (Group 2); and 1,251 individuals without Lyme borreliosis (Group 3). All samples were tested for B. burgdorferi B31 and B. afzelii antigens using ELISA. The positive results of the ELISA B. burgdorferi B31 assay were confirmed withWestern blot for the same strain. In Group 3, 162 (12.9%) patients were ELISA positive for B. burgdorferi B31, while only 6 (0.6%) patients had IgG ELISA antibodies to B afzelii. Bands in WB were detected in 104 (8.4%) of the Group 3 subjects. The bands found to be most reliable for the identification of strain B. burgdorferi B31 by IgG WB were those representing the 93, 39, 34, and 23-kDa proteins. Our results show that serologic diagnosis of Lyme borreliosis is far from clearly established. To date, the only reliable criteria are clinical ones correlated with laboratory evidence.
Copyright 2000 Wiley-Liss, Inc.
The prevailing scientific view is that Lyme disease is spread to human beings only through the bites of certain species of ticks.
But sometimes Lyme strikes multiple members of the same family -- the parents and the children. Some of those families believe that the bacterium that causes the debilitating symptoms of Lyme can also be spread through sexual contact. Others believe Lyme disease can be transmitted to children during a mother's pregnancy.
Lyme disease infections of humans are a big problem, and it's growing bigger. According to the Centers for Disease Control and Prevention, bacteria carried in some species of small ticks is now spreading Lyme disease to an estimated 300,000 new patients a year in the United States.
For some of those patients, the effects of Lyme disease are devastating. Caitlin Carlson lives with her family in Gilberts, Ill.
"My body just gets so tired and ill, and you just feel sick," the teenager told us. "And you can't explain it. Some days you just want to lay up in bed and do nothing because I feel so sick. And I'm 18, and I should not feel like that."
Caitlin Carlson believes her case of Lyme disease was passed on to her at birth from her mother, who has also been diagnosed with Lyme disease. Her father has also been diagnosed with the illness.
Read the rest of the story and watch the video:
http://www.myfoxdc.com/story/25646147/can-lyme-disease-be-spread-through-sex
Neurological scientists at Rush University Medical Center have found that using cinnamon, a common food spice and flavoring material, can reverse the biomechanical, cellular and anatomical changes that occur in the brains of mice with Parkinson's disease (PD). The results of the study were recently published in the June 20 issue of the Journal of Neuroimmune Pharmacology.
Read the whole story:
